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	<title>PharmaFeed &#187; Aberdeen University</title>
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		<title>Why Alzheimer’s patients tend to develop epilepsy</title>
		<link>http://www.pharmafeed.com/why-alzheimer%e2%80%99s-patients-tend-to-develop-epilepsy</link>
		<comments>http://www.pharmafeed.com/why-alzheimer%e2%80%99s-patients-tend-to-develop-epilepsy#comments</comments>
		<pubDate>Fri, 20 Mar 2009 11:59:49 +0000</pubDate>
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				<category><![CDATA[Alzheimers]]></category>
		<category><![CDATA[Aberdeen University]]></category>
		<category><![CDATA[Alzheimer’s]]></category>
		<category><![CDATA[beta-amyloid]]></category>
		<category><![CDATA[dementia]]></category>
		<category><![CDATA[Epilepsy]]></category>
		<category><![CDATA[Professor Tibor Harkany]]></category>

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		<description><![CDATA[An international team of scientists have found out why Alzheimer’s disease sufferers go on to develop epilepsy.
Led by scientists at Aberdeen University in Scotland, the research team have discovered that a protein in the brain accumulates in clumps in the brains of people suffering from dementia.
The clumps of proteins make the nerve cells too sensitive, [...]]]></description>
			<content:encoded><![CDATA[<p style="text-align: justify;">An international team of scientists have found out why Alzheimer’s disease sufferers go on to develop epilepsy.</p>
<p style="text-align: justify;">Led by scientists at Aberdeen University in Scotland, the research team have discovered that a protein in the brain accumulates in clumps in the brains of people suffering from dementia.</p>
<p style="text-align: justify;">The clumps of proteins make the nerve cells too sensitive, which in turn lose their ability to communicate coherently with other nerve cells.</p>
<p style="text-align: justify;">The researchers say that this makes Alzheimer’s patients more susceptible to seizures, reports the Scotsman.</p>
<p style="text-align: justify;">While official figures show that nearly one third of Alzheimer’s patients suffer from some degree of epilepsy, this is for the time that a link has been established between the two conditions.</p>
<p style="text-align: justify;">Professor Tibor Harkany, the Aberdeen neurobiologist who led the research, said that the findings could lead to changes in the drugs used to treat Alzheimer’s disease.</p>
<p style="text-align: justify;">He said that the research team had discovered that the “beta-amyloid protein”, a key component of the plaques that clog the brain of an Alzheimer’s patient, was causing cells to short-circuit and fire too many electrical signals.</p>
<p style="text-align: justify;">Source: Silver Scorpio- International News</p>
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